In gastric cancer, interleukins (IL-2 and IL-4) and TNF-α were enhanced by polysaccharides that also provide dose-dependent protection against N-methyl-N'-nitro-N-nitrosoguanidine (MNNG) induced oxidative injury by enhancing Superoxide dismutase (SOD), catalase (CAT), and glutathione (GSH-Px) [22]. The gene discussed is CAT; the disease is gastric cancer.