The addition of probiotics to this dysbiotic diet altered the presence of the different bacteria family/genus, reversed the loss of the integrity of the intestinal barrier and the plasmatic concentrations of LPS, attenuated the activation of the inflammatory pathway (NF-kB), increased the activation of the enzyme involved in a salvaged pathway (Akt), and finally resulted in a reduction of myocardial infarction size. This evidence concerns the gene NFKB1 and myocardial infarction.