FPR2 and metabolic dysfunction-associated steatotic liver disease: Furthermore, a recent experimental animal study found that Formyl Peptide Receptor 2 (FPR2) expression is higher in female mice than that in male mice, making females more resistant to the development and progression of NAFLD, and the severe damage seen in FPR2-depleted females supports FPR2’s protective role in female mice’s liver (49).