In contrast, in renal cell carcinoma, overexpression of miR-155-5p favors cell proliferation, colony formation, migration, and metastasis by regulating N-cadherin and Zinc finger protein SNAI1 (Snail) (35), associating increased expression with invasive phenotypes and metastasis (Figure 1). Tumor developed a unique metabolic pattern involved in all of its invasive behaviors. This evidence concerns the gene SNAI1 and hereditary clear cell renal cell carcinoma.