Recent study was reported that a selected group of autophagy-related genes, such as CCL2, cyclin-dependent kinase inhibitor 1A (CDKN1A), FOS, myelocytomatosis (MYC), and TNF superfamily member 10 (TNFSF10)—whose functions are related to IFN-I signaling pathways—significantly influenced the infiltration of multiple immune cells, including B-cells, macrophages, and NK cells, in samples from DM patients compared to controls, suggesting that these genes may be potential diagnostic biomarkers for DM (158). This evidence concerns the gene CCL2 and dermatomyositis.