In their study, Wei et al. (2021) observed that KIF2C was overexpressed in HCC and was related to several aggressive malignancies that activate the Wnt/β-catenin signaling pathway and was also involved in the HCC progression as it interacted with TBC1D7 in mTORC1 signaling. This evidence concerns the gene TBC1D7 and hepatocellular carcinoma.