Compared to the other three groups, every protein level and mRNA level of phosph-STAT1, the targets expression levels in the IFNγ and STAT3 inhibitor combination group were the highest in NSCLC cells (H1299, H1975, PC9, and H3255) (Figures 5(a)–5(d)), suggesting that inhibition of STAT3 phosphorylation may further activate the STAT1 phosphorylation level. This evidence concerns the gene STAT1 and non-small cell lung carcinoma.