When myocardial cell injury occurs, the cell membrane rupture leads to the troponin complex released from the myocardium into the blood, resulting in the high expression of cTnT in patients with sepsis, which also suggests that the abnormally high expression of cTnT is an important manifestation of impaired myocardial function and may have adverse effects on the prognosis of patients with sepsis [15, 16]. The gene discussed is TNNT2; the disease is Sepsis.