We also found 6 LP/P variants in 4 patients that we did not deem to be explanatory for the DCM, including 2 compound heterozygous truncating variants in CEP135, a de novo deletion of chromosome 14q22.3q23.1 (Hg19: 57,007,506-61,613,506), 2 compound heterozygous pathogenic missense variants in SLC37A4, and a de novo missense variant in MAP3K7. Here, SLC37A4 is linked to familial dilated cardiomyopathy.