The interference of Dectin-1 interaction with β-glucans by a soluble dectin-Fc fusion protein dampened the expression of inflammatory cytokines, TNFα, IL-1, IL-6, MIP-2, CCL3, G-CSF, and GM-CSF, expression in vivo, and increased fungal burden during aspergillosis (33). Here, CSF2 is linked to aspergillosis.