CLEC7A and candidiasis: Dectin-1 promoted the survival of antigen-specific CD4+ T cells, not the CD8+ T cells, specifically in GI-associated lymphoid tissues following systemic Candida infection, and ablation of Dectin-1 reduced the tissue-specific dendritic cells and increased activation of CD4+ T cells leading to higher susceptibility to Candida-induced colitis (35).