As a recent example of virus‐induced severe acute respiratory syndrome coronavirus 2 (SARS‐CoV‐2) infection, it is involved into defective type I interferon (IFN) activity and cytokine storms characterized by high levels of interleukin (IL)‐1β, IL‐6, IL‐12, and tumor necrosis factor (TNF), subsequently leading to acute respiratory distress syndrome (ARDS).12, 13. This evidence concerns the gene IFNA1 and acute respiratory distress syndrome.