Given the confounding effects of tumor heterogeneityin the clinical setting, we examined the robustness of our SSTR2-targetedFGS strategy using frozen sections from freshly resected pNETs (withadjacent normal tissues), metastatic lesions (liver), and lymph nodes.After confirming tumor histology with H&E staining and SSTR2 expressionby IHC, we incubated adjacent tissue sections with MMC(FNIR-Tag)-TOCand obtained fluorescence readouts using the Odyssey NIR imager. This evidence concerns the gene SSTR2 and neoplasm.