Our experiments here show that AML cells exposed to OG86 250 nM or DMSO vehicle for 48 h undergo the expected upregulation of myeloid differentiation markers CD11b and CD86, and that this is unaffected by induced expression of either the DNA-binding domain of GFI1 (GFI1-ZNF) or HMG20B alone. The gene discussed is HMG20B; the disease is acute myeloid leukemia.