The differentiation reprogramming of TNBC-basal cells carrying SNAI1 knockout correlated with FOXA1 upregulation (Fig. 6, Supplementary Fig. 4) and the lower expression levels of FOXA1 in basal tumors coupled with its negative correlation with SNAI1 (Fig. 6B, C, Supplementary Fig. 4A–E), agree with the proposed tumor suppression function of FOXA1 in breast cancer cells [67]. The gene discussed is FOXA1; the disease is breast cancer.