Both male and female patients who died of COVID-19 displayed significantly higher CRP concentrations compared to survivors.19 There seems to be a vigorous proinflammatory cytokine and chemokine response to the virus leading to apoptosis of endothelial cells, and epithelial cells which damages the pulmonary microvascular barriers and causes vascular leakage and alveolar edema, leading to ARDS. Here, CRP is linked to acute respiratory distress syndrome.