SRC and idiopathic pulmonary fibrosis: Forced overexpression of SRC in IPF-ABCs (SRC+ IPF-ABC) resulted in enhanced fibrosis and cellular remodeling in the alveolar compartment of NRG mouse lungs, whereas SRC knockdown in IPF-ABC cells (SRC− IPF-ABC) from the same donor caused markedly reduced remodeling (Fig. 4i).