Furthermore, sepsis led to a significant increase in plasma (Figures 2A,B)and myocardial (Figures 2C,D,G,H) levels of pro-inflammatory factors, such as TNF-α and IL-1β, compared with those in the control group, whereas the EA pre-treatment reduced inflammation compared with the LPS group (Figures 2A–F). The gene discussed is TNF; the disease is Sepsis.