The relationships between inflammation and atherosclerosis have been well demonstrated in RA: proinflammatory cytokines such as TNFα, IL-1 and IL-6 produced by various activated cells (T lymphocytes, monocytes, mastocytes, adipocytes) are released into the circulation and have potential effects on different tissues, including the blood vessels, leading to endothelial activation, vascular dysfunction as well as altered lipid profile and prothrombotic effects (6). This evidence concerns the gene IL6 and rheumatoid arthritis.