BRCA1 and breast carcinoma: The most enriched pathways were DNA double−strand break repair by homologous recombination (four genes), DNA double−strand break repair by non−homologous end joining (four genes), hereditary breast cancer signaling (six genes), role of BRCA1 in DNA damage response (five genes), DNA double−strand break repair by homologous recombination (three genes), and role of CHK proteins in cell cycle checkpoint control (three genes) (Supplementary Figure S4).