This has appeal as an area of study as it is one of the core processing deficits in schizophrenia, and one with elements that bridge symptom domains.8 It lends itself to a combined behavioural–neuroimaging approach with relative specificity, although of course administration of oxytocin may also affect related and potentially confounding processes such as emotion processing, mentalising and attribution. This evidence concerns the gene OXT and schizophrenia.