In various pathogenic conditions, IL-17A can clear the infection and initiate an inflammatory reaction that could be mediated through the NF-κB pathway,9 production of many inflammatory cytokines such as TNF-α, IL-1β,10 G-CSF, and IL-6 as well as chemotaxis of macrophages and neutrophils to the inflamed tissues.6 This evidence concerns the gene IL6 and infection.