ADGRL3 and attention deficit-hyperactivity disorder: Protein–protein interaction analyses using different protein–protein docking computational tools predicted a good molecular alignment between ADGRL3 and GIP within the first rank positions and docking scores (Table 2S), suggesting that this interaction may represent a vital ADHD etiological factor that could modulate neuroplasticity, neurotransmission, and cell proliferation during embryogenesis83–86, a significant event related to ADHD, and suggest that GIP receptors play an essential role in cognition, neurotransmission, and cell proliferation.