Park7 has been shown to facilitate liver progenitor cell expansion in a 3,5‐diethoxycarbonyl‐1,4‐dihydrocollidine diet‐induced liver injury murine model19 and promote the development of diethylnitrosamine‐induced hepatocellular carcinoma20; Park7 deficiency could improve carbon tetrachloride‐induced liver fibrosis and liver ischemia–reperfusion injury.18, 21. Here, PARK7 is linked to injury.