Mouse models of non-lethal malaria have been used to confirm the importance of inflammatory responses in contributing to the control of iRBCs, including the contribution of CD8 T cells which can recognize P. yoelii XNL-infected erythroblasts that express peptide-loaded MHC-I on their surface [309] and kill them via Fas-FasL dependent cytotoxicity [310] and ganulysin-mediated mechanisms of cytotoxicity [48]. Here, CD8A is linked to malaria.