The viscosity of β-glucan (in drink mode) could account for 79–96% of the changes in the plasma response of glucose and insulin to 50 g of glucose [103], delaying intestinal glucose absorption as it is trapped in micelles with β-glucan, attenuating the postprandial insulin response (in portal vein), and consequently decreasing the activity of the liver enzyme HMGCoA; these phenomena have beneficial effects in the control and prevention of type 2 diabetes and cholesterol synthesis [88,91]. Here, INS is linked to type 2 diabetes mellitus.