Nevertheless, together with our use of more than minimum sample size necessary for statistical evaluation and with fewer serotonin receptors found in alpha cells of type 2 diabetes patients via immunostaining [8], our result supports the notion that alpha cells may have a weaker response to the change in glucose level with the lack of serotonin stimulation, leading to the variable release of glucagon as seen in type 2 diabetes. The gene discussed is GCG; the disease is type 2 diabetes mellitus.