Contrarily, both α-MG (Figure 3D) and pterostilbene [14] increase phosphorylation of AKT and suppress periFN assembly on suspended tumor cells, legitimately reasoning why tumor cells treated with these two compounds in suspension are resistant to anoikis, a special type of cell apoptosis that occurs due to AKT inactivation and a loss of cell attachment [37]. The gene discussed is AKT1; the disease is neoplasm.