TNFRSF11B and deep vein thrombosis: Moreover, plaque calcification—a lesion associated with coronary thrombosis, even in the absence of eroded or ruptured plaque—is directly linked to the imbalance between the receptor activator of the nuclear factor NFκB ligand (RANKL) and osteoprotegerin (OPG), which are also expressed in endothelial cells [50,51,52].