The hormonal effect has a clear role and influence on the metabolic pathways at the adipose tissue level known up to now in tumor growth, such as the presence of high levels of leptin, giving a possible sequence that explains the oncogenic pathways dependent on the increase in circulating leptin that stimulate the secretion of proinflammatory cytokines, along with the overactivation of adipocytes [27]. This evidence concerns the gene LEP and neoplasm.