The presence of IFNγ induces the secretion of IP-10 and MIG, mainly by monocytes, plasmacytoid dendritic cells, endothelial cells and fibroblasts, recruiting to the site of infection IFNγ-producing lymphocytes such as Th1, Tc, NK and NKT cells, favoring their differentiation and proliferation, in addition to attracting more monocytes, macrophages and dendritic cells. This evidence concerns the gene CXCL10 and infection.