The presence of IFNγ induces the secretion of IP-10 and MIG, mainly by monocytes, plasmacytoid dendritic cells, endothelial cells and fibroblasts, recruiting to the site of infection IFNγ-producing lymphocytes such as Th1, Tc, NK and NKT cells, favoring their differentiation and proliferation, in addition to attracting more monocytes, macrophages and dendritic cells. The gene discussed is IFNG; the disease is infection.