There was a small degree of overlap between the potential markers derived from luminal, basal, or Her2-positive marker ‘seeds’, most likely indicating a degree of commonality involved in transcription, regulation and biological pathways between the three distinct breast cancer types explored, but many of the predicted marker genes remained limited to one of the three separate breast cancer types tested, summarized in Figure 8. This evidence concerns the gene ERBB2 and breast carcinoma.