In the group with retrochorial hematoma, plasminogen activity (%Plg) decreased significantly (Table 2, Figure 1C), indicating an abnormality of fibrinolysis that may be explained by the active consumption of plasminogen by the forming hematoma, or by the influence of other factors such as PAI-1, alpha-2-antiplasmin, etc. At the same time, there was no significant change in fibrinogen (Table 2), but there was a significant increase in SFMC (Table 2, Figure 1B). This evidence concerns the gene SERPINE1 and hematoma.