When the injury is cleared from phagocytosing dead cells and matrix debris, the process is followed by the termination of leukocyte infiltration and their apoptosis in a wound area as well as the secretion of anti-inflammatory molecules such as interleukin-10 (IL-10) and transforming growth factor-β (TGF-β) by macrophages and lymphocytes, which is required for proper transition to the next step and is crucial to avoid left ventricular dilatation [32,33]. This evidence concerns the gene IL10 and Left ventricular dilatation.