The main stimulated mechanisms are: (i) proliferation, growth, and survival of neoplastic cells (EGF family, IL-6, TGFβ) [72,73,74]; (ii) epithelial mesenchymal transition (EMT) and metastasis (TGFβ) [74,75]; (iii) angiogenesis (VEGF, FGF, CXCL8/IL-8) [76,77]; (iv) migration of immune system cells in the tumor site (CCL family, CXCL family) [78]; (v) immunosuppression (VEGF, IL-6, IL-10, TGFβ) [79]; (vi) stemness (KIT ligand, R-spondins) [80,81]; and (vi) ascitic fluid formation (VEGF) [82,83]. Here, CXCL8 is linked to neoplasm.