Cluster B had higher expression levels of ACSL4, ADIPOR1, CAMKK2, CPT1A, CPT1B, JAK2, NFKBIE, RELA, STAT3, TNFRSF1A and TNFRSF1B and lower levels of PRKAB2 and PRKCQ than that in cluster A. The same trend could be found between the epilepsy group and control group, indicating a more pronounced abnormal activation of the adipocytokine pathway in cluster B. As prior research discussed [33], adipocytokines were closely connected to many aspects of immunity and inflammation. The gene discussed is PRKAB2; the disease is epilepsy.