Dehydroabietic acid reduced hypertrophy by activating the Keap1/Nrf2-ARE signaling pathway, increasing the expression of the ferroptosis suppressor protein 1 (FSP1) gene, and inhibiting the accumulation of ROS to ameliorate non-alcoholic fatty liver disease [52]. The gene discussed is AIFM2; the disease is metabolic dysfunction-associated steatotic liver disease.