PPARA and Crohn disease: In both healthy controls and Crohn’s disease patients, exposure of Th17 cells to UCB left unchanged the levels of peroxisome proliferator-activated receptors α (PPARα) and γ (PPARγ) (Supplementary Fig. 6a, b) that were previously linked to control of glycolysis24 and inhibition of glycolytic genes like PGK125.