Comparing the miR expressions in renal biopsies of lupus nephritis, it was identified that miR-150 was related to higher chronicity level (chronicity index [CI] ≥4), suggesting as biomarker of specific histologic manifestations of lupus nephritis.60 Furthermore, miR-183 could mediate the TGF-β1/Smad pathway, in mice with lupus nephritis (LN) and in human renal glomerular endothelial cells (HRGECs).61 Here, TGFB1 is linked to lupus nephritis.