In addition to IL-6, the function of which in the pathogenesis of RA is well documented [28], the majority of these proteins, such as IL-7, IL-12B, TNFSF14, chemokines, and MMP-1, are likely to promote the inflammatory and destructive processes in RA [1, 2]. The gene discussed is TNFSF14; the disease is rheumatoid arthritis.