In multiple myeloma, the interaction of lncRNA protein disulfide isomerase family, a member 3 pseudogene 1 (PDIA3P) with c-Myc can not only enhance its reverse transcriptional activity and promote binding to glucose-6-phosphate dehydrogenase (G6PD) promoter and stimulate G6PD expression but also activate the pentose phosphate pathway (PPP), promoting tumor growth and drug resistance [37]. This evidence concerns the gene G6PD and AL amyloidosis.