Since PPA subtypes are associated with typical cognitive, MRI, and neuropathological findings, i.e., FTLD-tau with progressive supranuclear palsy (PSP) linked to prominent speech abnormalities with agrammatism and FTLD-TDP type C consistently associated with svPPA [18], specific clinical and MRI features should prompt consideration of an atypical neuropathological diagnosis [20]: DLB has been associated with lvPPA and little to no speech output during the day but with concurrent sleep talking [21] or svPPA with recurrent unprovoked visual hallucinations [22]. The gene discussed is MAPT; the disease is primary progressive aphasia.