In addition, the study found that the BCAT1 transcript was specifically associated with musashi RNA binding protein 2 (MSI2), a member of the Musashi RNA-binding protein family, and the binding of both parties can regulate the translation of BCAT1 and the activation of mammalian target of rapamycin 1 (mTORC1), which affects the MSI2-BCAT1 axis in the growth and development of CML (41). This evidence concerns the gene BCAT1 and chronic myelogenous leukemia, BCR-ABL1 positive.