AXL and Wilson disease: MET, AXL, KIT, and IGF1R were found overexpressed in WD/DDLPS cells versus normal adipocytes and pre-adipocytes (73) and genetic analyses evidenced common amplifications of IGF2, IGF1R, ERBB3 together with FGFR1, FGFR3 and PDGFR in WD/DDLPS (74–76).