Moreover, the pathways of cell adhesion molecules, chemokine signalling pathway, autoimmune thyroid disease, type I diabetes mellitus, primary immunodeficiency, and graft versus host diseases were enriched in KIRC and LUAD (Figures 9(a) and 9(b)), and the signalling pathway of asthma, allograft rejection, virtual myocarditis, the internal immune network for IgA production, and Staphylococcus aureus infection were enriched in LUCS and READ (Figures 9(c) and 9(f)). This evidence concerns the gene CD79A and graft versus host disease.