These data support our findings, as we observed that the CCL-2 molecule is increased at T0 in the AKI(+) group, as it also occurs with CXCL-8 and IL-6 molecules (Figure 2), which are already described as elevated after Bothrops envenomation, thus influencing the increase in the acute inflammatory response in these patients. This evidence concerns the gene CXCL8 and acute kidney injury.