SRSF3 and myocardial infarction: Thus, findings from our group and others suggest that in addition to targeting SRSF3/p21 to mediate the senescence of CMFs, miR‐486 also has the potential to target cardiomyocytes and endothelial cells in the myocardium via multiple pathways to exert beneficial effects on the survival of cardiomyocytes and cardiac angiogenesis in ischaemic myocardium and facilitate the regeneration of MI.