These findings suggest that capsaicin might act as a cocarcinogen in a TRPV1‐independent but EGFR‐dependent manner.[52] Furthermore, capsaicin has also been reported to boost the proliferation and survival of androgen‐responsive prostate cancer LNCaP cells, which corresponds to enhanced androgen receptor expression.[53]. The gene discussed is EGFR; the disease is Familial prostate cancer.