Other miRNAs that have emerged to undergo AR control, thus affecting cell growth and cellular morphology in BC are miR-21, whose androgen-induced reduction is a result of the recruitment of HDAC3 at MIR21 gene promoter by AR (58), miR-100 and miR-125, which are inversely correlated with DHT-induced matrix metalloproteinase MMP13 (57), and miR-328-3p that increases upon DHT administration in MDA-MB-231 cells, acting as one of the mechanisms by which DHT reduces CD44 protein levels and limits cell motility and adhesion (59). This evidence concerns the gene AR and breast cancer.