Among the results of our preliminary experiments, the elevated HIF-1α levels on days 2 and 3 post-MI verified the hypoxic cardiac microenvironment, while the upregulation of TGF-β1 and OPN1 proved the occurrence of inflammatory reactions and cardiac repair [33, 34] and was consistent with Dewald et al. [35] and Deten et al. [36]. This evidence concerns the gene HIF1A and myocardial infarction.