MT can induce a G0/G1 cycle arrest in human non-small cell lung cancer cells by downregulating the activity of the epidermal growth factor receptor (EGFR)-Akt signaling pathway and inhibiting the growth of human non-small cell lung cancer cells; MT can inhibit tumor volume in vivo and weight, with inhibition rates of tumor transplanted mice were 16.29 and 35.35%, respectively (Li et al., 2018). This evidence concerns the gene AKT1 and non-small cell lung carcinoma.